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Microbiota and immune responses at weaning predict the susceptibility to chronic...

Microbiota and immune responses at weaning predict the susceptibility to chronic inflammatory diseases in adulthood Microbiota dysbiosis is associated with chronic inflammatory diseases such as allergy, inflammatory bowel diseases, cancer and metabolism-related disorders. It has been reported that exposure to microbial and dietary components ea... see more

31/08/2026

UNIVERSITAET BERN

2M€

Project Budget: 2M€

Project leader

UNIVERSITAET BERN No se ha especificado una descripción o un objeto social para esta compañía.

TRL 4-5

PARTICIPATION DEPralty Sin fecha límite de participación.

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Financing granted El organismo H2020 notifico la concesión del proyecto The day 2021-07-14

ERC-2020-STG: ERC STARTING GRANTS Scope:Objectives

I+D

Cerrada does 5 years

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characteristics of the participant

Este proyecto no cuenta con búsquedas de partenariado abiertas en este momento.

Private Information

No hay información privada compartida para este proyecto. Habla con el coordinador.

2 Participantes

UNIVERSITAET BERN

1.56M€ | Leader

BIOMET SPAIN ORTHOPAEDICS,S....

489.72K€ | participant

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Project duration: 61 months Date Start: 2021-07-14
End date: 2026-08-31

Project leader

UNIVERSITAET BERN No se ha especificado una descripción o un objeto social para esta compañía.

TRL 4-5

Project Budget 2M€

participation deadline Sin fecha límite de participación.

Project description Microbiota dysbiosis is associated with chronic inflammatory diseases such as allergy, inflammatory bowel diseases, cancer and metabolism-related disorders. It has been reported that exposure to microbial and dietary components early in life may program the immune system to develop tolerance or susceptibility to chronic inflammatory diseases with age. We have recently shown that vigorous immune response induced by gut microbiota at weaning, prevents the development and exacerbation of chronic inflammatory diseases in adult mice. Such immune response is termed the weaning reaction. Antibiotic exposure and/or excessive fats intake during this critical window dysregulate the weaning reaction and increase the subsequent susceptibility to develop immunopathology. However, our current knowledge of the biological processes underlying the weaning reaction remains largely unknown. Here we aim to determine the biological processes of the weaning reaction and define the microbial and nutritional signatures at weaning that predict the susceptibility to adult pathology. These aims will be assessed using high-throughput sequencing, machine-learning algorithms and axenic and gnotobiotic mice models. Our project will help deciphering how nutrition and microbiota at weaning orchestrate the immune system development and impact adult chronic inflammatory pathologies susceptibility.

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